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Variability in supplementation dose and duration, concentration of bioactive compounds, participant characteristics, exercise tests and statistical considerations may help explain these disparate findings

Similarly, HDAC3 also represses genes involved in DNA damage response and cell cycle checkpoints, enabling AML cells to evade genotoxic stress induced by chemotherapy ( In summary, dysregulated histone deacetylation establishes an epigenetic environment conducive to AML progression and therapeutic resistance

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