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In the past decades, many NSAIDs have been correlated with gastrointestinal problems, liver injuries and dyspepsia (34- 47)

122 Interestingly, research has shown that in the early stages of hypoxia-induced human and mouse PASMCs, the 3-untranslated region of miR-17/20a directly targeted and primarily inhibited PHD2, leading to HIF-1 degradation to promote VEGF expression, and subsequently enhances proliferation

VTE1 interacted strongly with OsJ_36874 and ARSM2 , consistent with its catalytic role in DMPBQ formation (Fig

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