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Furthermore, the in vivo fate of assembled nanostructures and potential immunogenicity from peptide or nucleic acid-based modules warrant careful consideration [270,271,272,273,274]

a , Total GPX4 activity of HT1080 cells overexpressing GFP-GPX4 WT , GFP-GPX4 K48A , or GFP-GPX4 K48L and a control line

Sun Z, Schwenzer A, Rupp T, Murdamoothoo D, Vegliante R, Lefebvre O, Klein A, Hussenet T, Orend G

In this context, pro-inflammatory cytokines (e.g., TNF, IL-1, IL-8) can activate NET formation ( In the context of CVD, NETs not only exert cytotoxic and prothrombotic effects (Warnatsch et al., 2015), but also contribute to plaque formation (Silvestre-Roig et al., 2019)

Multidimensional investigation of potential anti-cancer targets of glutamine metabolism and their inhibitors Targeting glutamine metabolism-related enzymes and their inhibitors As indicated by the aforementioned evidence, it can be hypothesised that glutamine-addicted cancer cells are reliant on GLS for survival

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