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Mitochondrial dysfunction and oxidative damage to sarcomeric proteins

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Therefore, current advanced strategies focus on up-armoring these therapeutic cells to withstand TME-induced oxidative stress, primarily by enhancing their intrinsic antioxidant capacity and mitochondrial fitness

Preclinical hypogonadism in genetic hemochromatosis in the early stage of the disease: evidence of hypothalamic dysfunction

There is also evidence that elevated homocysteine may be linked to uric acid 4 levels

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