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Zulazmi et al

The term inflamm-aging is used to describe this age-related immune phenotype of inflammation, characterized by a chronic, low-grade pro-inflammatory state, which is related to both activation of the innate immune system and continued exposure to antigens and stress ( Increased cell ageing and exposure to endogenous infectious agents and harmful signals throughout life imbalance the innate and adaptive immune responses, shifting toward greater involvement of innate immune cells, to the detriment of adaptive immunity based on antigen response ( With ageing, there is an alteration of the adaptive immune response affecting T and B lymphocytes, decreasing the ability to respond to antigens (Roubenoff et al., 1998

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PMID: 32 LinXShaoTWenXWangMWenCHeZ

Faust HJ, Cheng T-Y, Korsunsky I, Watts GFM, Gal-Oz ST, Trim WV, Kongthong S, Jonsson AH, Simmons DP, Zhang F, Padera R, Chubinskaya S, Albrecht J, Anolik JH, Apruzzese W, Barnas JL, Bathon JM, Ben-Artzi A, Boyce BF, Boyle DL, Bridges SL, Bykerk VP, Campbell D, Ceponis A, Chicoine A, Curtis M, Deane KD, DiCarlo E, Donlin LT, Dunn P, Filer A, Carr H, Firestein GS, Forbess L, Geraldino-Pardilla L, Goodman SM, Gravallese EM, Rao D, Gregersen PK, Guthridge JM, Gutierrez-Arcelus M, Holers VM, Horowitz D, Hughes LB, Ivashkiv LB, Ishigaki K, James JA, Kang JB, Keras G, Lakhanpal A, Lederer JA, Lewis MJ, Li Y, Liao K, Mandelin AM, Mantel I, Marks KE, Maybury M, McDavid A, McGeachy MJ, Mears JR, Meednu N, Millard N, Moreland L, Nayar S, Nerviani A, Orange DE, Perlman H, Pitzalis C, Rangel-Moreno J, Raza K, Reshef Y, Ritchlin C, Rivellese F, Robinson WH, Rumker L, Sahbudin I, Sakaue S, Seifert JA, Scheel-Toellner D, Singaraju A, Slowikowski K, Smith M, Tabechian D, Utz PJ, Weinand K, Weisenfeld D, Weisman MH, Xiao Q, Zhu Z, Li ZJ, Cordle A, Wyse A, Wei K, Raychaudhuri S, Lynch L, Moody DB, Brenner MB, Accelerating Medicines Partnership RASLEN

Two working peptides, a few carriers, and nothing added for the label