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doi: 10.1080/10409238.2019.1570075 102 KangH.OkaS.LeeD

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This may result from tumor-mediated glucose restriction, which limits T-cell substrate availability and suppresses mTOR activity, glycolysis, and IFN- production, thereby promoting functional exhaustion while promoting tumor progression [171]

Integrating these datasets with machine learning and systems biology approaches will enable the construction of predictive models that classify patients according to metabolic vulnerabilities, thereby guiding rational trial design and therapeutic personalization