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It is precisely due to distinctions in EC metabolism during Phase II (glucuronidation, sulfation, methylation) and its conversion capacity by the gut microbiota that variations arise in its in vivo exposure levels and the composition of EC and its active metabolites

Davis-Smyth, T., Duncan, R

3.1.6 CEACAM6 CEACAM6 exhibits differential expression across CRC molecular subtypes, with highest levels in CMS4 tumors characterized by stromal infiltration and poor prognosis