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Additionally, GPxs due to their subcellular compartmentalization (cytoplasmic, membrane, ER, and mitochondrial localizations) control the local concentration gradient of peroxides and thereby regulate the activation/deactivation of redox-responsive signaling pathways [1, 8,9,10]

In a rat model of DOCA-salt hypertension, vascular mitochondrial ROS production was markedly elevated, accompanied by the activation of other ROS sources

Preservation of these spaces may facilitate regenerative cells and growth factors infiltration and more sustained exposure of biologic agents and their signaling mediators, thereby supporting tissue remodeling

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In the blood of patients with SE, lower levels of SOD, catalase, GSH, and total antioxidant capacity were found (Pauletti et al., 2019)

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