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[1] Initial human trials showed that it retains the lipolytic properties of human growth hormone without stimulating IGF-1 production

Rat experiments demonstrated enhanced regeneration of uterine and testicular tissue following induced damage, with histology highlighting reduced fibrosis and enhanced vascular repair

TEV protease cleaves between Q (Gln) and G (Gly) or S (Ser)

ROS cause lipid peroxidation, DNA damage, and protein denaturation, which directly impair cell functions and activate pro-inflammatory signaling pathways (e.g., NF-B and Nrf2) (Muro et al., 2024), exacerbating inflammation and creating a vicious cycle between inflammation and oxidative stress

Masubuchi Y, Bourdi M, Reilly TP, et al

1 2 Clinical Orthopaedics and Related Research