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4 Hypothesis of the gut microbiota-ferroptosis axis in OA pathogenesis 4.1 Gut microbiota and abnormal iron metabolism synergistically promote joint ferroptosis 4.1.1 Gut microbiota promotes ferroptosis via hepcidin-FPN1 axis and FTN-mediated iron overload The gut microbiota modify systemic iron homeostasis through the modulating ability of microbiota to regulate the synthesis of hepatic hepcidin, an effect the subject of intense investigation

Elevated levels of extracellular heat-shock protein 72 (eHSP72) are positively correlated with insulin resistance in vivo and cause pancreatic -cell dysfunction and death in vitro

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doi: 10.1016/0168-583X(96)00125-5

Lineage tracing reveals the bipotency of SOX9 + hepatocytes during liver regeneration

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