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Furthermore, TMAO promotes diabetes by elevating fasting insulin, increasing insulin resistance (HOMA-IR), and inducing adipose tissue inflammation, contributing to glucose metabolism dysfunction and heightened cardiovascular event risks in diabetic patients (Subramaniam and Fletcher, 2018)

Mechanism and physiological role Its main function is to participate in the transport of fatty acids to the mitochondria and in metabolic adaptation during changes in demand (fasting, exercise or physiological stress)

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Recent studies demonstrate that prolonged supplementation, especially in combination with carbohydrates (CHO), may increase muscle total carnitine (TC) content in skeletal muscle [5,6,7]

For instance, the immunometabolism of T cells heavily relies on the continuous dynamic reshaping of mitochondria through fusion and fission events to maintain mitochondrial quality