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doi: 10.1371/journal.pone.0002698

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5.1 Iron chelators and iron modifiers Iron chelators and modifiers effectively inhibit cellular ferroptosis by regulating the expression and activity of iron transporters and storage proteins, including transferrin and its receptor (TfR1), ceruloplasmin, ferroportin, divalent metal transporter 1 (DMT1), and other membrane iron transport proteins (24, 26)

The assumption that antioxidants exhibit a lag phase and that the length of the lag phase is positively correlated with antioxidant capacity underpins the use of the lag phase

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Therefore, this study aims to summarize the recent progress on the role of ferroptosis in different kidney diseases to provide strong support for the treatment and prevention of these diseases ( Figure 3 )