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Clinical characteristics and mortality risk factors of these patients were analyzed

Thiol redox control via thioredoxin and glutaredoxin systems

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Inflammation is a key secondary pathological process in spinal cord injury, and KD attenuates inflammation, including reduced nuclear factor-kappa B pathway and expression levels of TNF-, IL-1, and interferon- (149)

By contrast, in the GSH-C4-first sequence (Figure 3B), HA + GSH-C4 elicited a reproducible cytokine activation profile: at V3, levels of IFN-2 (p = 0.044), IFN- (p = 0.023), TNF- (p = 0.039), IL-6 (p = 0.044), IL-12p70 (p = 0.015), IL-23 (p = 0.034), and the epithelial alarmin IL-33 (p = 0.023) were significantly elevated versus baseline, whereas IL-1, MCP-1, IL-8, IL-10, and IL-18 remained unchanged

NAC treatment also significantly upregulated the expression of TNC and TNMD proteins, which were abrogated by inhibitor of the PI3K/AKT signaling in the NAC group of TSPCs in vitro