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Potent antioxidant function at the expense of central carbon metabolism The dependency of NRF2-addicted cancer cells on exogenous glutamine is attributable to the elevated consumption and excretion of glutamate as a result of two major transcriptional outputs of NRF2 (Fig

Supporting nutrients: Glutathione synthesis requires not just cysteine (from NAC) but also glutamate and glycine

57 This study suggests the feasibility of targeting BCAAs metabolism in TME mesenchymal and cancer cells for PDAC therapy (Fig

Different pathways participate in ferroptosis induction and defense

GSH is a key antioxidant associated with the regeneration of ascorbic acid (AsA) in the AsAGSH cycle, which is an important pathway for clearance of reactive oxygen species (ROS) under kinds of stress ( 2 O 2 to H 2 O, thereby reducing or eliminating the toxicity of H 2 O 2

Hanging gondola structure of the T1 domain in a voltage-gated K + channel