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Since excessive vascular ROS generation is the key driver of endothelial dysfunction and the main trigger of eNOS uncoupling, attempts have been made to mitigate oxidative stress

Antioxidant therapy for hypertension: a mini-review on the recent findings

The oxidative milieu not only damages cellular components but also sensitizes hepatic tissue to inflammatory stimuli, fostering activation of Kupffer cells and the release of proinflammatory cytokines such as TNF-, IL-6, and IL-1