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A reduction in glycolytic progress was detected in erastin-induced ferroptosis, which might be caused by increased synthesis of ATP and oxidative phosphorylation (OXPHOS)[10, 115]

Bioenergetics and the formation of mitochondrial reactive oxygen species

Stem cells, endothelial cells, and various repair cells can migrate more effectively toward damaged tissue through a process called chemotaxis

In naturally aged mice and in doxorubicin-treated mice, FOXO4-DRI treatment significantly improved physical fitness, restored fur density, improved renal function, and improved liver function effects attributed to the selective elimination of accumulated senescent cells

To further verify if the effects of H 2 O 2 treatment on mitochondrial proteostasis and mitophagy were indeed mediated through cysteine deprivation and CLPXP complex inactivation, the three cancer cell lines were treated simultaneously with both H 2 O 2 and cysteine (H 2 O 2 +Cys) or H 2 O 2 , ZK53 and CLPP overexpression (H 2 O 2 + ZK53 + CLPP OE)

An overview of the ferroptosis hallmarks in Friedreichs ataxia