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In conclusion, the altered redox state of cancer cells represents a potential target for ROS-manipulating anticancer therapies

Summary Keywords GHK, TGF-1, pulmonary fibrosis, epithelial-to-mesenchymal transition, Smad, collagen Citation Zhou X-M, Wang G-L, Wang X-B, Liu L, Zhang Q, Yin Y, Wang Q-Y, Kang J and Hou G (2017) GHK Peptide Inhibits Bleomycin-Induced Pulmonary Fibrosis in Mice by Suppressing TGF1/Smad-Mediated Epithelial-to-Mesenchymal Transition

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A variety of pharmacological approaches have been tried to limit cognitive symptoms and other aspects of secondary injury following mTBI