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As the executor of inflammatory cell death, GSDMD forms transmembrane pores that can trigger mitochondrial-dependent oxidative stress or release pro-inflammatory mediators, such as interleukin-1 (IL-1) and high-mobility group box 1 (HMGB1) 6,7,8,9,10,11,12

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In a variation, the CDRs are initially obtained from mRNA (or cDNA) from antibody-producing cells (e.g., plasma cells/splenocytes from an immunized wild-type mouse, a human, or a transgenic mouse capable of making a human antibody as in WO 92/03918, WO 93/12227, and WO 94/25585), including hybridomas derived therefrom