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Analgesic effects of clinically used compounds in novel mouse models of polyneuropathy induced by oxaliplatin and cisplatin

Quite recently, experiments conducted using mice kidney and tubular cells showed that cisplatin-induced kidney inflammation might be governed by the activation of poly(ADP-ribose) polymerase-1 (PARP-1), an enzyme which in the hyperactivated state decreases the rate of glycolysis and impairs electron transport and ATP formation, which eventually leads to dysfunction and/or death of various normal cell types

J Anus Rectum Colon

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FT-iron levels in the striatum rise significantly, while TfR levels decrease, and FPN levels increase to manage the excess iron (Bartzokis et al., 2007

Ranasinghe SL, Fischer K, Zhang W, Gobert GN, McManus DP