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E.LuisierR.TahaD

BNIP3 induction by hypoxia stimulates FASN-dependent free fatty acid production enhancing therapeutic potential of umbilical cord blood-derived human mesenchymal stem cells

These changes are supported by fundamental metabolic reprogramming that enables neurons to meet increasing energy and biosynthetic demands

Consequently, there is an increased influx of FFAs into the liver, driven both by reduced inhibition of lipolysis and by an increase in de novo synthesis of FFAs [49]

Here, it binds to antioxidant-responsive elements, initiating the transcription of various antioxidant genes [such as NAD(P)H:quinone oxidoreductase 1, glutathione S-transferase, heme oxygenase 1, -glutamylcysteine ligase and GSH] (58)