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The cellular uptake mechanism of HMPB-H@M1EV and HMPB-H was investigated by treating the cells with various endocytic pathway inhibitors using flow cytometry (Supplementary Fig

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In subgroups of patients in which NAC was the only antioxidant given, mortality was significantly reduced in at the longest follow-up time

Copper excess triggers oxidative stress (by impairing antioxidant enzymes and NO bioavailability) and disrupts Ca homeostasis, leading to endothelial dysfunction, cardiac hypertrophy, and contractile impairment

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