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Disease states of GH excess (e.g., acromegaly) and GH deficiency (GHD) (e.g., congenital isolated GHD) are characterized by increased and decreased GH and IGF-I levels, where the GH/IGF-I relationship is reflected by a primary association (high GH and high IGF-I for acromegaly and low GH and low IGF-I for GHD)

At the same time, SIRT1 deacetylates multiple transcription factors, such as Nrf2, promotes its entry into the nucleus and activates the expression of antioxidant stress genes, such as HO-1 and GPX4, the activation of which helps mitigate cell damage caused by ferroptosis

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Electrophysiological recordings demonstrate that Dihexa-treated neurons exhibit significantly increased frequency of miniature excitatory postsynaptic currents (mEPSCs), indicating enhanced synaptic transmission