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The classical PINK1/Parkin pathway recruits effectors, including ubiquitin-sensing autophagy receptors (e.g., OPTN, NDP52) and LC3, to the MAMs membrane and lipid environment, where they tag and segregate damaged mitochondria [101, 102]

Neurochem Res 8: 231243

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These findings suggest Dihexa not only prevents synaptic loss but actively promotes synapse formation even in the presence of Alzheimer's pathology