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The typical clinical manifestation is bowel obstruction in a critically ill patient becoming febrile, with signs of acute cholecystitis, dehydration, and aspecific biochemical abnormalities such as leukocytosis, elevated aminotransferase levels, electrolyte imbalance due to dehydration, and concomitant heart, pulmonary, and metabolic comorbidities [179]

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This then provokes an immune response from the T and B cells of the immune system and the production of antibodies against tissue transglutaminase (Lionetti 2011

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Mutations in MT-TQ, MT-TH, MT-TK, MT-TS1, MT-ND1, MT-ND5, MT-ND6 and MT-TS2 are also associated with MELAS syndrome

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