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In addition, Cu deficiency can lead to mitochondrial dysfunction by inducing the expression of other mitochondria-associated molecules ( The main regulator of cellular redox balance is the transcription factor NF-E2-related factor 2 (Nrf2), which inhibits oxidative stress, inflammation, and apoptosis (Zhong et al., 2022)

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When excessive LPS is transferred to the systemic circulation and increases plasma LPS levels, it can trigger inflammation, thereby facilitating the onset and progression of sarcopenia (Thoma and Lightfoot, 2018)