Lipid accumulation in L02 cells after FFA treatment could cause increased ROS, mitochondrial damage, and ferroptosis
Medical treatments deliver additional glutathione through glutathione injection or intravenous drips
School of Education & Human Sciences Emily Horr, Holton, Kansas
J Surg Res 164:e99e105 Yamamoto M, Kensler TW, Motohashi H (2018) The KEAP1-NRF2 system: a thiol-based sensor-effector apparatus for maintaining redox homeostasis
Paul Summa cum laude: Helena Aklog, Teoman Aktan, Rafael Alvarez Tarrios, Olivia Anderson, Esther Anderson, Jakaea Andrews, Maria Armada, Sabrina Armada, Kaleab Ashenafi, Quincy Aust, Calum Axelson, Jon Bailey-Zimmerman, Hazel Bandsma, Bailey Beedle, Wyatt Bennig, Thea Berg, Isaac Bergstrom, Josephine Black, Ava Bleifuss, Daniel Blumer-LaMotte, Caleb Bole, John Bradford, Benjamin Brunner, Quinn Buchl, Satoshi Callahan, Robert Carlson, Pablo Contreras-Galeana, Christopher Crutchfield, Flynn Cunningham, William Davies, Mason Deegan, Benjamin Drexler Booth, Claire Duffy, Aili Earl-Torniainen, Julian Falk, Cormac Flynn, Calla Fragassi, Jonatha Franquiz, Clara Gaw, Nina Gorgeson, Naod Getachew, Hannah Gliedman, Gabriel Gonzalez, Violet Grant, Ava Griffin, Miriam Harrleson, Ashley Harris, Liam Hefferan, Amy Hernandez Grande, Caelyn Hippen, Violet Hirman, Evan Hoglund, Elsa Houge, Amelie Isom, Steian Johnson, Isaac Johnson, Sophie Johnson, Morgan Johnson, August Johnson, Miles Kaker, Beatrice Kealey, Corinne Kipfmueller, Mary Koch, Hanna Koch, Lauren Kottke, Henri Kukkonen, Liya Lantyderu, Donovan Lee, Grace Lewis-Mosher, Grace Little, Savannah Lofstrom
found that DIBI, a polymer hydroxypyridinone iron chelator, reduces ocular inflammation in endotoxin-induced uveitis by decreasing local and systemic endotoxin-induced ocular inflammation
