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AD is characterized by the accumulation and aggregation of misfolded proteins in brain, the sustained activation of the UPR mediated by the misfold proteins initiates or mediates neurodegeneration (Hoozemans et al., 2005)

In contrast, loss of p53 prevents the accumulation of dipeptidyl-peptidase-4 (DPP4) in the nucleus, triggering membrane-associated DPP4-mediated lipid peroxidation

To efficiently combat rising MGD in agricultural systems, significantly more efforts are required to match soil providing capacities with crop demand by precise plant and soil testing, proper fertilizer formulation, and optimal fertilization rates and timing to minimize net soil mining (Figure 7)

The review also explores protective effects of ApoE mutations against AD and ApoE4-induced tauopathy, neurodegeneration, and neuroinflammation

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