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Furthermore, loss of amylo-1,6-glucosidase (AGL), a glycogen debranching enzyme, leads to elevated SHMT2 levels, which in turn increases glycine and purine nucleotide synthesis and further supports tumor cell proliferation (78)

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These molecular features make the cells sensitive to corresponding inhibitors, and preclinical research on the application of HER2/EGFR inhibitors, CDK inhibitors, and MAPK inhibitors in suppressing brain metastatic cells is underway