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They are promising molecular targets for drug discovery to alleviate the onset of diseases and solve nonresponses and partial responses as well as adverse effects for current DMARDs

[14] Both variants are believed to stem from a strongly protective effect against Plasmodium falciparum and Plasmodium vivax malaria

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demonstrated that erastin-induced ferroptosis elevated lysosome-associated membrane protein 2a (LAMP2a) expression and promoted chaperone-mediated autophagy (CMA) while facilitating GPX4 degradation, suggesting a possible connection between autophagy and ferroptosis (Wu et al., 2019)