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A recent report with amylin receptor structures clearly showed that the N-terminal part of amylin interacted with the TM domain of the amylin receptor and that the C-terminal part of amylin interacts with amylin receptor ECD [15]

A 2025 systematic review reported 7 of 12 maintained improvement beyond 6 months

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doi: 10.1523/jneurosci.3203-08.2008 59 GuanJ

It may be argued that the atrophy and malfunction of the SV is a significant contributor to ARHL, since the loss of EP plays a significant role in the rise of the hearing threshold in mammals