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Compared with diabetic rats, the left ventricular hemodynamic parameters and myocardial ultrastructure changes (myocardial hypertrophy and fibrosis) are obviously normalized in diabetic animals treated with NaHS, which may be associated with upregulation of cardiac myosin light chain kinase (MLCK) and downregulation of serum creatine kinase MB isozyme (CK-MB) (Yang et al., 2016a)

Studies have suggested that the uptake of HMCD complexes, including MHI-148-HSA and genistein-IR-783 conjugates, could be mediated by micropinocytosis and lipid-raft endocytosis ( Figure 2 ) (58, 74)

Each tag requires a specific cofactor (vitamin, mineral, or amino acid) to be available

J Biol Chem 277: 11581165

doi: 10.1200/JCO.2017.77.5841 125 MateusAMatssonPArturssonP

Inflammation in atherosclerosis: pathophysiology and mechanisms