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Highly pathogenic coronavirus N protein aggravates lung injury by MASP-2-mediated complement over-activation

For example, AI-driven methods for multi-omics integration can better capture disease heterogeneity and inform clinical decision-making [642, 643]

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Elevated ROS increases PRDX2 monomer formation, which causes TIMELESS-TIPIN dissociation from the replisome and replication slowdown, thereby inducing replication stress

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