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Stroke and Excitotoxicity Protection Excitotoxicity protection appeared in stroke models where bilateral carotid artery clamping produces ischemia-reperfusion injury with massive glutamate release

Along with its known function combating oxidative stress, GSH is also important for the detoxification of xenobiotics and metabolites, the regulation of cell growth and cell death, the regulation of the nitric oxide cycle, the storage of cysteine, the maintenance of redox potential and the modulation of immune function

There is a great deal of evidence suggesting that DNA methylation defects are associated with ASDs, and the role of the methylenetetrahydrofolate reductase (MTHFR) gene in folate metabolism may contribute to epigenetic mechanisms that modify complex gene expression, thus causing autism

These results are in line with the previous studies, which similarly demonstrated Spirulinas hypolipidemic activity and its potential role in lipid metabolism [58,59,60,61]

126 Hepatic necrosis may be centrilobular (zone 3) or panlobular (Figure 61-20 )

4bd), accompanied by impaired muscle function and glucose metabolism (Fig