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doi: 10.1016/j.immuni.2019.09.020

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In a demyelination mouse model with neuroinflammation, KD treatment inhibits the activation of pro-inflammatory glial cells and decreases the production of pro-inflammatory cytokines, including IL-1 and TNF-, in addition to significantly reducing histone deacetylase 3 and NLRP3 (151)

Thus, our findings are likely the result of the potent antioxidant capacity of GSH facilitating the engagement of accumbal circuitry to produce motivated behavior, as well as GSH-independent mechanisms involving more complex and long-term modifications, including a reshaping of synaptic glutamatergic activity

This series is not about dosing recommendations

Oxidative damage has been linked to high levels of eryptosis through an increase in the intracellular free Ca 2+ concentration, as evidenced by in vitro studies [24, 134, 300, 301] in patients intoxicated with lead [302], and in diabetic patients with chronic renal damage [303]