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It is believed that additional regulated forms of non-apoptotic cell death likely remain to be discovered that mediate cell death in other developmental or pathological circumstances

Research on mixture effects involving multiple EEDs remains an emerging field, with most studies focusing on single-chemical exposures despite the reality of multi-chemical environmental exposure, limiting our understanding of cumulative health risks ( Transgenerational Impacts Lack Adequate Human Documentation Animal studies consistently demonstrate that EED exposure can induce heritable epigenetic modifications affecting offspring fertility across multiple generations

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338 However, in heart failure, DEAD-box helicase 17 and B-cell lymphoma 6 are downregulated, which would reverse the inhibition of Drp1 and promote mitochondrial fission

10.1039/c8fo00654g 31 CunhaA

RNA-seq revealed differential gene expression in oxidative phosphorylation, glutathione metabolism, and detoxification processes, suggesting the involvement of antioxidant defence in SalB-mediated neuroprotection