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Genetics, environmental triggers, and infections contribute to this misdirected immunity

[18] See 21 C.F.R

All the above operations were performed according to the manufacturers instructions

In addition to non-HLA Abs induced by an alloimmune mechanism, Abs directed to self-antigens, such as angiotensin II type 1 receptor (AT1R), perlecan, endothelin type A receptor (ETAR), vimentin, or Rho GDP-dissociation inhibitor 2 (ARHGDIB), have been found to be associated with inferior graft outcome, although their causal role in the pathogenesis of damage is unclear (29, 30)