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Disulfidptosis may be an important pathological mechanism in neurodegenerative diseases

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recently identified the diphenylbutene derivative compound 3f as a ferroptosis inhibitor, and they showed that this compound can protect against ischemic stroke in rats by increasing FSP1 protein levels, 233 suggesting that novel agents that upregulate FSP1 may be used to treat ferroptosis-related diseases

doi: 10.1002/ddrr.106 81

The expression levels of glial fibrillary acidic protein (GFAP) and ionized calcium-binding adapter molecule 1 (Iba-1), which usually serve as markers of astrocytes and microglia, were elevated in the MFeI MNs-treated mice to levels similar to those of healthy mice (control group) (Fig

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