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However, under defined pathophysiological stresses, including inflammatory cues, metal overload, cysteine deprivation, or drug exposure, GSH oxidation and thiol chemistry may become coupled to metal redox cycling and mitochondrial damage amplification, thereby increasing susceptibility to forms of regulated cell death such as ferroptosis and cuproptosis

Ruan, Q., Wang, C., Zhang, Y

fumigatu s cell extracts (3) (Figure S5), however, GliT-mediated gliotoxin reductase activity is detectable upon addition of gliotoxin (4)

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CGI-58 resembles lipases structurally

Tang XH, Knudsen B, Bemis D, Tickoo S, Gudas LJ