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10.1097/00042737-199910000-00002 108 UysJ

(2016b) revealed that Tanshinone IIA mitigated the overactivity of glycogen synthase kinase (GSK)3, as well as the hyperactivation of its downstream mitogen-activated protein kinases, which are fundamentally involved in renal fibrogenesis and inflammatory processes

TF is continuously delivered to the clot to participate in its growth [13]

Inhibition of serine metabolism can lead to YAP degradation, resulting in TBK1 activation and increased production of type I IFN and related cytokines [137]