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The metabolic reprogramming of the mitochondrial network in tumoral programs is achieved through several mechanisms, including ncRNAs transcribed both in the nuclear and in the mitochondrial genome (mtDNA)

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Taken together, metabolomic studies of long COVID, ME/CFS, and MDD show considerable similarities in the overall metabolic profiles associated with a disruption of energy production and an imbalance of potentially neuroprotective and detrimental metabolites, i.e., a deficiency of neuroprotective metabolites such as acetylcarnitine

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More importantly, the supplementation of acetate could counter ROS-mediated EC dysfunction in CPT1A-deficient mice, providing therapeutic opportunities in related disorders