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J Clin Endocrinol Metab 90, 678-88 (2005)

There is considerable evidence that many of the biochemical pathways adversely affected by hyperglycemia including glucose oxidation, the formation of advanced glycation end-products (AGE), and activation of polyol pathways, are associated with the generation of reactive oxygen species (ROS), ultimately leading to increased oxidative stress in a variety of tissues [3]