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Oleuropein protects mitochondria by mitigating mitochondrial superoxide production [121]

In this work, we demonstrate that deletion of the pHAT domain induces a more pronounced disruption in O-GlcNAc feedback homoeostasis, suggesting increased catalytic activity or broader substrate promiscuity (Supplementary Fig

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Palmitate-induced lipotoxicity is crucial for the pathogenesis of nonalcoholic fatty liver disease in cooperation with gut-derived endotoxin