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People with fully functional immune systems are also at risk for life-altering fungal infections such as fungal keratitis, a common cause of infection-related vision loss, or chronic aspergillosis-associated asthma 1,2

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While numerous substantial challenges remain, in vivo neurochemistry has already built a crucial bridge, connecting invasive work in animals, perception and the biochemistry of human visual function

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the AGE-RAGE signaling, which induces axonal atrophy through p38 MAPK activation (Sango et al., 2025), and the PKC- activation, which impairs nerve blood flow via endothelin-1 overexpression ( Hyperglycemia-induced mitochondrial dysfunction in neuronal and Schwann cells leads to excessive O 2 production at complex I and III of the ETC, which initiates a cascade of injury through four primary mechanisms (Wu L

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