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doi: 10.3389/fneur.2018.00713 58 BianchiVELocatelliVRizziL

Acta 1 , 1939 (2003)

Research suggests that the SARS-CoV-2 virus can directly hijack mitochondrial function during the acute infection phase, altering the mitochondrial membrane potential and downregulating the expression of key metabolic enzymes

Additionally, excessive ROS leads to oxidative damage to DNA and proteins, as well as lipid peroxidation of cell membranes, ultimately resulting in cell death (21)

Ferroptosis is an iron-dependent, lipid peroxidation-driven form of programmed cell death, the execution of which centers on the uncontrolled accumulation of PL-OOH (phospholipid hydroperoxides), and unlike the accidental necrosis caused by severe external insults, ferroptosis is a form of programmed cell death