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This senolytic approach, addressing DNA damage-induced senescence, offers a promising strategy for eradicating residual disease and improving cancer treatment outcomes

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*Correspondence: Marie McIlroy, [email protected] This article was submitted to Molecular and Cellular Oncology, a section of the journal Frontiers in Cell and Developmental Biology Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers

GSK-3 also plays a role in APAP-induced liver injury, since silencing GSK-3 was shown to attenuate JNK activation and inhibit APAP hepatotoxicity65

& Risbridger, G

However, the limitations of the study included retrospective study design, missing data, and distance of patient residence from the monitoring centers