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The preservation of NAD+ through NNMT inhibition has profound implications for cellular metabolism

The knockout mouse data is particularly useful for interpreting this: long-term AOD-9604 treatment in beta3-AR-deficient mice failed to produce body weight reduction or sustained lipolysis, confirming that the chronic fat loss effect depends on this receptor pathway

This compound is attracting attention for its ability to influence fat cell biology, energy metabolism, and even markers of healthy aging without relying on appetite suppression or stimulant-based mechanisms

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May help regulate blood sugar levels, reducing the risk of metabolic disorders

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