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It is well known that anorexigenic proopiomelanocortin (POMC) neurons function to the hypothalamic paraventricular nucleus (PVN) and ventromedial nucleus of the hypothalamus (VMN), where alpha-melanocyte-stimulating hormone (-MSH) is produced, by activating the melanocortin-4 receptor (MC4R)

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[16] [17] Whereas the latter directly act on the dopamine transporter (DAT) to inhibit the reuptake and/or induce the release of dopamine, bromantane instead acts via indirect genomic mechanisms to produce a rapid, pronounced, and long-lasting upregulation in a variety of brain regions of the expression of tyrosine hydroxylase (TH) and aromatic L-amino acid decarboxylase (AAAD) (also known as DOPA decarboxylase), key enzymes in the dopamine biosynthesis pathway

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