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One possibility would be combining GHK with antioxidants, such as the mitochondrial targeted antioxidant SS31 [23], given the role of redox imbalance in persistent lung fibrosis [15]

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Importantly, in the two trials that used supplemental vitamin D3, the risk reduction was apparent among participants with a BMI below the median of 31.3 kg/m2, but not among those whose BMI was 31.3 or higher

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The presence of IgG3 has also been found to predict the treatment response to IVIg, which is thought to work at least partly by saturating FcRn and thereby preventing endogenous IgG from being salvaged from degradation [163, 164]

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