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In the other form of plasticity, increased morphological proximity to synapses occurred in one sub-population, while increases in extra-synaptic GLT1 expression occurred in another sub-population of cells (Kruyer et al., 2022)

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Selanks interactions with these neurotransmitters may at least partially explain its anxiolytic effects [14]

However, the study showed that BDNF-TrkB signaling plays a necessary role in mediating CSDS-induced social avoidance behavior in the ventral tegmental area (VTA)-NAc, and established BDNF-TrkB signaling as a pathological mechanism during chronic stress [116]

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