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in this compound acts as a modulator of hepatic cytochrome P450 enzymes (Lin et al., 2022)

Glutathione disulfide (GSSG) was recovered in the diets and digesta from the LGSH and HGSH group, which could demonstrate the auto-oxidation of GSH

The overproduction of RNS in activated macrophages, leukocytes, and epithelial cells in the intestinal mucosa is caused by iNOS, which is only found in inflammatory tissue (136)

The capacity to metabolize xenobiotics has made development of drugs very time consuming and costly due in large part to: Interindividual variations in the capacity of humans to metabolize drugs Drug-drug interactions Metabolic activation of chemicals to toxic and carcinogenic derivatives Species differences in expression of enzymes that metabolize drugs, thereby limiting the use of animal models to predict effects in humans Most xenobiotics are subjected to 1 or multiple enzymatic pathways that constitute phase 1 oxidation and phase 2 conjugation